Black potato disease

Symptoms mainly infect stems or tubers, and may develop from the seedling stage to the later stage of reproduction. Seed disease rots into lumps, does not germinate, or just germinates and rots in the soil and cannot sprout. Seedlings infected with common plant heights of 15 to 18 cm appeared with symptoms, plants were short, internode shortening, or leaf roll up, chlorotic chlorosis, or black belly, wilting and death. The three main vascular bundles turn brown in the transverse stem. Tuber disease begins at the umbilicus and expands radially toward the pith. The disease is dark brown. The vascular bundle is also dark brown when viewed across the vascular bundle. The skin is not separated by pressing with the hand. When the humidity is high, the tuber turns dark brown and rots. Stinking, different from bacterial wilt.

Pathogen Erwinia carotovora subsp. atroseptica (Van Hall) Dye said that Carrot, Erwinia carotovora, a potato subspecies, is a low-temperature type that causes soft rot in the genus Erwinia. Bacterial short rod-shaped, single-celled, rarely double-connected, flagellate week, with a British film, size 1.3-1.90.53-0.6 (μm), Gram-negative, can ferment glucose production gas, colony micro convex Milky white, round and round edges, translucent and reflective, thick and viscous. The bacteria suitable temperature 10 to 38 °C, the most suitable for 25-27 °C, higher than 45 °C loss of vitality.

Transmission routes and disease conditions Seed tuber fungus, the soil generally does not carry bacteria; bacteria first spread through the cut potato block infection, causing more species of potato disease, and then enter the plant through the vascular bundle or pith, causing the upper part of the disease. Field pathogens can also be transmitted through irrigation water, rain, or insects, and they can be invaded by wounds. Pathogens in the later stages of the disease are transmitted from the stems of the ground through the stems to the newly growing tubers. During the storage period, pathogenic bacteria are infected by the diseased sweet potato and penetrated into the wound or lenticels to infect the potato. Ventilation in the cellar is poor or the humidity is high and the temperature is high, which is conducive to the expansion of the disease. The rate of colonization was high or the number of rainy or low-lying plots was heavy.

Control methods (1) Selection of disease-resistant varieties such as Yifang No.1, Shengli No.1, Anti-dii No.2, Xinhui No.2, Xinhui No.4 and Xinshu No.2. (2) The disease-free seed potato was selected to establish a disease-free field, and (3) the cutting was planted immediately after planting with plant ash. (4) timely early sowing, promote early emergence. (5) The diseased plants were found to be excavated in time. In particular, it was necessary to carefully remove the field and reduce the source of bacteria. (6) The seed potatoes must be carefully selected before entering the pit. After entering the pit, the management is strengthened. The pit temperature is controlled at 1-4°C to prevent the cellar temperature from being too high and the humidity to be too high.

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